3 edition of Experimental leukemogenesis. found in the catalog.
|Statement||Edited by Tadashi Yamamoto and Haruo Sugano.|
|Series||GANN monograph on cancer research,, no. 12|
|Contributions||Yamamoto, Tadashi, 1917- ed., Sugano, Haruo, 1925- ed., Nihon Gan Gakkai.|
|LC Classifications||RC643 .J36 1970|
|The Physical Object|
|Pagination||viii, 321 p.|
|Number of Pages||321|
|LC Control Number||72002137|
Mertens, D. and S. Stilgenbauer (). "Ibrutinib-resistant CLL: unwanted and unwonted!" Blood (11): , IF ; C.C. Oakes, M. Seifert, Y. Assenov, L. Although they comprise only a small proportion of the nucleated cell population within the marrow, HSCs are responsible for the daily production of more than 10 11 red cells, granulocytes, and platelets in mice, HSCs account for % of all marrow accrue within the trabecular marrow in endosteal and subendosteal regions 8, 9, circumscribing the vasculature and Cited by: 4.
Experimental Hematology Vol November , Pages Comparative utility of NRG and NRGS mice for the study of normal hematopoiesis, leukemogenesis, and therapeutic responseCited by: 4. Canonical Wnt signaling is critically involved in normal hematopoietic development and the self-renewal process of hematopoietic stem cells (HSCs). Deregulation of this pathway haCited by:
In the present article, we review the experimental and clinical evidence for a role of β3 integrins for leukemogenesis and chemosensitivity in AML. Next Article in Journal Gallic Acid Alleviates Hypertriglyceridemia and Fat Accumulation via Modulating Glycolysis and Lipolysis Pathways in Perirenal Adipose Tissues of Rats Fed a High-Fructose DietCited by: 9. The PAF complex synergizes with MLL fusion proteins at HOX loci to promote leukemogenesis Andrew G. Muntean, 1 Jiaying Tan, 1 Kajal Sitwala, 1 Yongsheng Huang, 2 Joel Bronstein, 1 James A. Connelly, 1 Venkatesha Basrur, 1 Kojo S. J. Cited by:
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Experimental leukemogenesis. [Baltimore] University Park Press  (OCoLC) Online version: Japanese Cancer Association Symposium on Experimental Leukemia Research in Japan ( Tokyo). Experimental leukemogenesis.
[Baltimore] University Park Press  (OCoLC) Material Type: Conference publication: Document Type: Book. The present reviews illustrate that chemokines can be involved in leukemogenesis. The chemokine system is also important both for the crosstalk between malignant cells and their neighbouring nonmalignant stromal cells (including endothelial cells) as well as for immunoregulation in patients treated with allogeneic stem cell : Hardcover.
The induction of preleukemia represents the initial phase in leukemogenesis. A sequence of further events (that could differ qualitatively from the initiating factor) is often required for the completion of the neo-plastic by: 7.
Book Description: The world of experimental leukemia research is a miniature of Experimental leukemogenesis. book research. Cancer research started from experimental leukemia/sarcoma research of Ellermann and Bang and Peyton Rous in early s.
Experimental leukemia includes most of important elements of cancer research; both viral and chemical carcinogenesis; most of. The present reviews illustrate that chemokines can be involved in leukemogenesis. The chemokine system is also important both for the crosstalk between malignant cells and their neighbouring nonmalignant stromal cells (including endothelial cells) as well as Experimental leukemogenesis.
book immunoregulation in patients treated with allogeneic stem cell transplantation. Cytogenetics of Preleukemic Stages in Experimental and Human Leukemogenesis.
Authors; Authors and affiliations This process is experimental and the keywords may be updated as the learning algorithm improves. Hameister H., Eul J. () Cytogenetics of Preleukemic Stages in Experimental and Human Leukemogenesis.
In: Grundmann E., Beck L Author: H. Seidel, F. Carbonell, H. Hameister, J. Eul. In the context of leukemogenesis, aberrant CpG island methylation in promoter regions, for example of tumor suppressor genes such as cyclin-dependent kinase inhibitor 2B (CDKN2B), which encodes p15 INK4b and CDKN2A, which encodes p16 INK4a and p14 ARF, is a well described phenomenon, and is associated with transcriptional silencing, which also involves recruitment of methyl-binding proteins Cited by: Experimental Hematology Today - presents the latest results of research reflecting the diverse interests of basic and clinical hematologists.
The major areas explored are hematopoietic regulation by cytokines; hematopoietic cellular growth regulation, with emphasis on the interaction of stromal with hematopoietic stem and progenitor cells; granulopoietic regulators; gene transfers into. Leukemias are monoclonal diseases that arise from cells in the hematopoietic stem and progenitor cell compartment.
Consistent with emerging models of carcinogenesis, leukemogenesis is an evolutionary process that involves multiple independent genetic and epigenetic by: Stephen J. Forman, MD Chair, Department of Hematology and Hematopoietic Cell Transplantation City of Hope Comprehensive Cancer Center Duarte, CA, USA.
Robert S. Negrin, MD Professor of Medicine Chief, Division of Blood and Marrow Transplantation Stanford University Stanford, CA, USA. Joseph H. Antin, MD Professor of Medicine, Harvard Medical School Chief, Stem Cell.
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Similar books to The Chemokine System in Experimental and Clinical Hematology (Current Topics in Microbiology and Immunology Book ). Twenty-three major papers, and a few minor ones, on various currently important topics in viral, radiation, and chemical leukemogenesis, all originally presented at a.
Experimental Hematology Today - presents the latest results of research reflecting the diverse interests of basic and clinical hematologists. The major areas explored are hematopoietic regulation by cytokines; hematopoietic cellular growth regulation, with emphasis on the interaction of.
Implications of hemopoietic progenitor cell kinetics and experimental leukemogenesis: Relevance to Gompertzean mortality as possible hematotoxicological endpoint Yoko.
The active mutation of ras genes is a known additional hit in MLL-rearranged leukemia , , , and was so in our model. The hematopoietic cells undergoing the first hit must survive until the additional hit occurs during by: Abstract.
The majority of experimental radiation leukemia studies have been concerned with thymic lymphomas whereas only a few have been reported on radiation induced myeloid leukemia. The early systematic studies involved myeloid leukemia induction in the RF strain by exposure to whole body ionizing by: 7.
Hematopoiesis places clinical and experimental observations in the context of cell biology evaluates the effects of treatment, disease evolution, and the induced proliferative stress on the hemopoietic system concentrates on hemopoiesis within a clinical framework relating to immunology, cytogenetics, and leukemogenesis presents the.
Cancer Signaling: From Molecular Biology to Targeted Therapy 1st Edition Leibniz Institute for Experimental Virology in Hamburg, Germany.
She is a highly regarded expert in the field of leukemogenesis and hematology. Her areas of research are gene regulation, molecular control of differentiation, and hematopoietic stem cells.
5/5(1). Thank you for your interest in our work. Unfortunately, we currently have no open positions. However, we would like to invite you to apply for the outstanding programs at the DKFZ!. This book describes the Notch signaling pathway with a focus on molecular mechanisms.
The Notch signaling pathway is a seemingly simple pathway that does not involve any second messenger. Upon ligand binding two consecutive proteolytic cleavages of the NOTCH receptor release the Notch intracellular domain from the membrane.These findings provide strong evidence demonstrating that the S67 phosphorylation of MYC is associated with the tumor-promoting effect of AURKB and is functionally important for MYC stabilization and T cell leukemogenesis in vivo.
AURKB Inhibitor Synergizes with Vincristine to Induce T-ALL Cell DeathAuthor: Jue Jiang, Jingchao Wang, Ming Yue, Xiaolian Cai, Tianci Wang, Chao Wu, Hexiu Su, Yanwu Wang, Meng H.Abstract.
Benzene is a very common environmental contaminant. Due to its proven epidemiologic association with acute myelogenous leukemia (AML) in exposed workers, this aromatic chemical has been classified as one of the few known human carcinogens (EPA Class A, IARC Class I).
If human risks from background exposures to benzene are assessed according to EPA’s linearized multistage Author: Bruce Molholt.